By the end of this chapter you'll be able to…

  • 1State the three anatomical and physiological facts that reduce paediatric airway reserve
  • 2Recognise the classic history of an inhaled foreign body and its symptom-free interval
  • 3Explain the ball-valve mechanism and predict the radiographic findings
  • 4State why a normal chest radiograph does not exclude an inhaled foreign body
  • 5Apply the age-specific choking algorithm
  • 6Explain the electrolytic mechanism of button battery injury
  • 7Distinguish a button battery from a coin radiographically
  • 8State the timing of battery removal and the role and limits of honey or sucralfate
  • 9Recognise unilateral foul nasal discharge as a foreign body
  • 10Grade the severity of stridor before diagnosing its cause
  • 11Recognise the pre-arrest pattern of falling respiratory rate and quietening stridor
  • 12Distinguish croup, epiglottitis, bacterial tracheitis and retropharyngeal abscess
  • 13State the dose and rationale for dexamethasone and nebulised adrenaline in croup
  • 14State why a child given nebulised adrenaline must be observed
  • 15State the epidemiological effect of Hib vaccination on epiglottitis
  • 16List the actions contraindicated in suspected epiglottitis
  • 17Explain why retropharyngeal abscess is a disease of the under-fives
  • 18Distinguish primary from secondary post-tonsillectomy haemorrhage
  • 19Manage paediatric epistaxis and recognise its red flags
  • 20Explain why alkali and acid ingestion injure different organs
  • 21Recognise bilateral choanal atresia and manage it immediately
  • 22Describe the natural history of laryngomalacia
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Why this chapter matters in NEET PG
Paediatric ENT emergencies are not adult conditions occurring in smaller people. The airway is absolutely small with resistance rising as the fourth power of the radius, oxygen reserve is minimal, and children cannot report what happened. The result is that deterioration is abrupt rather than gradual and the calm-looking child is not necessarily safe. Clinically this chapter prevents a set of stereotyped disasters: a peanut left in a bronchus because the radiograph was normal, a button battery treated as a coin, a child with epiglottitis examined with a tongue depressor, and a quietening stridor read as improvement rather than exhaustion.

Pediatric ENT Emergencies

Paediatric ENT emergencies are not adult conditions occurring in smaller people. They behave differently because the anatomy and the physiology are different, and the difference is always in the same direction: less reserve.

Three facts generate almost every rule in this chapter.

The airway is absolutely small, and resistance rises with the fourth power of the radius. One millimetre of circumferential oedema in an infant trachea can raise resistance sixteen-fold. The same millimetre in an adult barely registers.

Oxygen reserve is minimal. Children consume oxygen at roughly twice the adult rate per kilogram while holding a proportionally smaller functional residual capacity, so the interval between apnoea and desaturation is measured in seconds.

Children do not report symptoms reliably. A toddler will not describe when something was swallowed, so the history comes from a parent who may not have seen the event, and the physical findings are often the only evidence.

The clinical consequence is that paediatric airway problems deteriorate abruptly rather than gradually, and that the calm-looking child is not necessarily the safe one.

1. Inhaled Foreign Body

Peak incidence is in toddlers, who explore with the mouth and lack molars to grind food. Peanuts, seeds and small toy parts dominate.

The classic history is a sudden choking or coughing episode, often followed by a symptom-free interval that falsely reassures everyone.

The right main bronchus is the commoner destination because it is wider, shorter and more vertical than the left, an anatomical asymmetry that becomes more marked with age.

Reading the chest radiograph

Most inhaled objects in children are organic and therefore radiolucent, so the film shows the effect rather than the object.

A foreign body acts as a ball valve: air enters past it on inspiration when the airway widens, and is trapped on expiration when the airway narrows. The result is hyperinflation of the affected side, not collapse, at least initially.

The findings are therefore a hyperlucent lung, a depressed hemidiaphragm and mediastinal shift away from the affected side, best seen on an expiratory film or a lateral decubitus view.

A normal chest radiograph does not exclude an inhaled foreign body. This is the single most important sentence in the section. A convincing choking history is an indication for rigid bronchoscopy regardless of the film, because the alternative is a child who develops recurrent pneumonia, bronchiectasis or lung abscess over the following months.

Rigid bronchoscopy under general anaesthesia is both diagnostic and therapeutic, and is preferred to flexible bronchoscopy because it secures the airway while permitting instrumentation.

The child who is choking now

A child with an effective cough is encouraged to cough and is not interfered with, because any intervention risks converting a partial obstruction into a complete one.

Once the cough becomes ineffective, the sequence differs by age. In an infant under one year, give five back blows followed by five chest thrusts, alternating; abdominal thrusts are avoided because the liver is relatively large and unprotected by the rib cage.

In a child over one year, give five back blows followed by five abdominal thrusts. If the child becomes unresponsive, begin cardiopulmonary resuscitation, checking the mouth for a visible object before each set of breaths but never sweeping blindly.

2. Swallowed Foreign Body

Most swallowed objects that reach the stomach pass uneventfully. The oesophagus is where they lodge, at three natural narrowings, of which the cricopharyngeus is the commonest.

A button battery in the oesophagus is a different order of emergency, and it is the highest-yield fact in this chapter.

The mechanism is not chemical leakage but electrolysis. Current flows through mucosa, generating hydroxide ions at the negative pole, and the resulting alkali produces liquefactive necrosis. Injury begins within two hours.

FeatureCoinButton battery
Radiograph, frontUniform discDouble ring or halo sign
Radiograph, lateralUniform edgeStep-off at the edge
UrgencyRemove if symptomatic or lodgedRemove immediately, ideally within 2 hours

Removal takes priority over everything, and nothing should delay it. Current guidance permits honey, or sucralfate suspension, to be given while awaiting endoscopy in ingestions of up to 12 hours, since coating the battery slows local hydroxide generation. Honey is used in children aged 12 months and over.

Two caveats matter. Neither agent substitutes for removal; they slow damage rather than preventing it. And neither is given if the battery may have been present for more than 12 hours, or if perforation is suspected.

Late complications include oesophageal stricture, tracheo-oesophageal fistula and, catastrophically, aorto-oesophageal fistula, which can present with sentinel bleeding days after removal.

3. Foreign Bodies in the Nose and Ear

Unilateral foul-smelling nasal discharge in a child is a foreign body until proved otherwise. No other diagnosis explains one-sided purulent rhinorrhoea in a well child so reliably.

Nasal foreign bodies risk posterior displacement and inhalation, so removal is attempted only with adequate immobilisation and equipment.

A nasal button battery is an emergency for the same electrolytic reason as an oesophageal one, and it can destroy the septum within hours.

Aural foreign bodies are less urgent unless they are batteries, vegetable matter that swells, or a live insect, which is killed with oil before removal to prevent damage from movement.

4. Stridor in the Emergency Department

The critical initial task is grading severity, not diagnosing cause.

A falling respiratory rate with quietening stridor in a tiring child indicates impending arrest, not improvement. Reduced air movement produces less noise. Agitation, then drowsiness, then a silent chest is the sequence to fear.

ConditionAgeOnsetKey features
Croup6 months to 3 yearsGradual, at nightBarking cough, hoarse voice, low fever, no drooling
Epiglottitis2 to 6 years, now rareRapid, hoursToxic, drooling, muffled voice, sitting forward, no cough
Bacterial tracheitisAnyFollows viral illnessToxic child, thick purulent secretions, poor steroid response
Retropharyngeal abscessUnder 5DaysNeck stiffness, refusal to move neck, odynophagia
Inhaled foreign bodyToddlerSuddenChoking history, unilateral signs

Croup

Laryngotracheobronchitis, usually parainfluenza, producing subglottic oedema, which is why the stridor is often biphasic and the cough barking.

A single dose of oral dexamethasone is the treatment, and guidelines commonly use 0.6 mg/kg, with evidence for lower doses of 0.15 to 0.3 mg/kg being less clear-cut. It works even in mild disease and reduces return visits and admission.

Nebulised adrenaline, around 400 micrograms per kilogram to a maximum of 5 mg, is added in moderate to severe disease. It improves symptoms within 30 minutes but wears off after about two hours, so a child who has received it must be observed rather than discharged on the improvement.

Epiglottitis

Once a disease of Haemophilus influenzae type b, it became rare after Hib vaccination, with reported incidence in under-fives falling by more than 90 per cent. It has not disappeared, and now occurs in unvaccinated children, in vaccine failures, and with other organisms.

Do not examine the throat, do not lie the child flat, do not attempt cannulation. Keep the child upright with a parent, call the most senior anaesthetist and ENT surgeon available, and inspect and intubate in theatre with a surgical airway prepared. Antibiotics follow airway control.

Retropharyngeal abscess

Occurs in children under about five, because the retropharyngeal lymph nodes that drain the nasopharynx involute after that age.

The child holds the neck stiffly, refuses to swallow, and may be misdiagnosed as meningitis. A lateral neck radiograph shows widening of the prevertebral soft tissue, and contrast CT defines the collection.

The risks are airway obstruction and downward spread into the mediastinum, and treatment is intravenous antibiotics with drainage of a significant collection.

5. Tonsils, Quinsy and Bleeding

Acute tonsillitis is usually viral. The Centor criteria, comprising fever, tonsillar exudate, tender anterior cervical nodes and absence of cough, estimate the likelihood of streptococcal infection.

Peritonsillar abscess, or quinsy, is a disease of older children and adults, presenting with severe unilateral pain, trismus, a muffled "hot potato" voice and deviation of the uvula away from the swelling. Treatment is drainage by aspiration or incision plus antibiotics.

Never give ampicillin or amoxicillin when infectious mononucleosis is possible, since it produces a florid maculopapular rash.

Post-tonsillectomy haemorrhage

The division by timing is the examinable point.

Primary haemorrhage occurs within 24 hours and is a surgical problem, usually requiring return to theatre.

Secondary haemorrhage occurs at around 5 to 10 days, is caused by infection and separation of the slough, and is usually managed with antibiotics and observation, though a significant bleed still needs theatre.

Any child who has swallowed blood may have a deceptively empty mouth, so tachycardia, pallor and repeated swallowing are more reliable than visible bleeding.

Corrosive ingestion

Accidental swallowing of household acid or alkali remains common in India, where cleaning agents are often decanted into drink bottles.

The two agents injure differently. Alkali causes liquefactive necrosis that penetrates deeply and continues after contact, so oesophageal injury is severe. Acid causes coagulative necrosis, forming an eschar that limits depth, but it damages the stomach more.

Three interventions are actively harmful and are the examinable point. Do not induce vomiting, which re-exposes the oesophagus. Do not attempt neutralisation, which is exothermic and adds a thermal burn. Do not pass a blind nasogastric tube.

Absence of oral burns does not exclude significant oesophageal injury, so endoscopy within the first 24 to 48 hours grades the damage and predicts stricture. Strictures develop over weeks and may need repeated dilatation for years.

6. The Neonatal Airway

Choanal atresia presents dramatically because neonates are obligate nasal breathers. Bilateral atresia produces cyclical cyanosis relieved by crying, since crying forces mouth breathing.

The diagnosis is suggested by failure to pass a catheter through each nostril and confirmed by CT. An oral airway stabilises the child. It is associated with CHARGE syndrome, so a murmur or a coloboma should be sought.

Laryngomalacia is the commonest cause of stridor in infants. The stridor is inspiratory, worse on feeding, crying and lying supine, and better when prone. It appears in the first weeks, peaks around six months and usually resolves by 18 to 24 months.

Most cases need only reassurance and growth monitoring. Failure to thrive, apnoea or severe obstruction indicates supraglottoplasty.

Subglottic stenosis is most often acquired, following prolonged neonatal intubation, and gives biphasic stridor with recurrent "croup" that does not behave like croup.

7. Epistaxis and Acute Ear Pain

Nosebleed in a child

Almost all paediatric epistaxis arises from Little's area on the anterior septum, where five vessels anastomose in the Kiesselbach plexus, immediately beneath thin mucosa that a finger can reach.

First aid is anatomical: pinch the soft cartilaginous part of the nose, not the bony bridge, for ten uninterrupted minutes, with the child sitting forward so blood is not swallowed.

Recurrent bleeds respond to silver nitrate cautery of the visible vessel and topical antiseptic cream. Never cauterise both sides of the septum at the same sitting, since bilateral mucosal injury risks septal perforation.

Two features change the assessment entirely. Recurrent heavy epistaxis with nasal obstruction in an adolescent boy suggests juvenile nasopharyngeal angiofibroma, which must not be biopsied. And bleeding from multiple sites, or with bruising, points to a coagulopathy rather than a local cause.

Acute mastoiditis in a child

Untreated or partially treated acute otitis media can progress to mastoid empyema, and the child presents with a tender postauricular swelling, loss of the postauricular sulcus and a pinna pushed forward and downward.

It needs admission, intravenous antibiotics and often cortical mastoidectomy, and it can progress to the same intracranial complications as cholesteatoma.

8. Worked Examples

Example 1. An 18-month-old choked while eating peanuts three days ago. He is now well with a normal examination and a normal chest radiograph. The parents want to go home. What do you advise?

Rigid bronchoscopy. A convincing choking history is an indication for bronchoscopy regardless of a normal radiograph, because most paediatric inhaled foreign bodies are organic and radiolucent, so the film can only show secondary effects. The symptom-free interval after the initial choking is expected and falsely reassuring. Left in place, a peanut causes recurrent pneumonia, bronchiectasis or lung abscess over subsequent months, and organic material swells and provokes intense inflammation, making later removal harder.

Example 2. A 3-year-old swallowed "a coin" two hours ago. The radiograph shows a disc in the upper oesophagus with a double ring appearance. What is it and what happens now?

A button battery, not a coin. The double ring or halo sign on the frontal film, and a step-off at the edge on the lateral, distinguish a battery from a coin. This is an emergency requiring endoscopic removal immediately, ideally within two hours of ingestion, because electrolysis generates hydroxide at the negative pole and produces liquefactive necrosis that begins within that window.

While arranging endoscopy, honey may be given since he is over 12 months and the ingestion is under 12 hours, to coat the battery and slow hydroxide generation, but it must not delay removal by a minute. Watch afterwards for stricture, tracheo-oesophageal fistula and the catastrophic aorto-oesophageal fistula.

Example 3. A 2-year-old has had a barking cough and stridor at night for two days, with a low fever, a hoarse voice and no drooling. He is alert and feeding. What is the diagnosis and treatment?

Croup, from subglottic viral oedema, usually parainfluenza. The barking cough with hoarseness and the absence of drooling or toxicity separate it from epiglottitis, in which the child is toxic, drooling, has a muffled rather than hoarse voice and characteristically has no cough.

Treatment is a single dose of oral dexamethasone, commonly 0.6 mg/kg, which works even in mild disease and reduces return visits. Nebulised adrenaline is added if he deteriorates, but its effect wears off after about two hours, so a child given adrenaline is observed rather than discharged on the improvement.

Example 4. A 4-year-old with stridor has become quieter over the last 20 minutes. His respiratory rate has fallen from 50 to 28 and he is drowsy. The nurse reports he is settling. Comment.

He is not settling; he is failing. A falling respiratory rate with quietening stridor in a child who was previously distressed means reduced air movement rather than reduced obstruction, and drowsiness indicates hypercapnia and exhaustion.

This is the immediate pre-arrest state. The correct response is emergency escalation to the most senior airway team available, preparation for intubation with a surgical airway option, and no attempt to examine the throat or reposition the child in a way that might precipitate complete obstruction.

Example 5. A newborn becomes cyanosed when quiet and pink when crying. A catheter cannot be passed through either nostril. What is the diagnosis, and what is the immediate management?

Bilateral choanal atresia. Neonates are obligate nasal breathers, so complete posterior nasal obstruction causes cyanosis at rest, relieved when crying forces the mouth open, producing the characteristic cyclical pattern.

The immediate management is to establish an oral airway, which bypasses the obstruction entirely, and to feed by tube until definitive surgery. Confirm with CT of the skull base. Look actively for CHARGE syndrome associations, including coloboma, heart defects, growth retardation, genital and ear anomalies, since choanal atresia is one of its defining features.

Summary

Paediatric airways have almost no reserve: resistance rises with the fourth power of radius, and desaturation follows apnoea within seconds.

A convincing choking history mandates rigid bronchoscopy regardless of a normal chest radiograph.

Inhaled foreign bodies favour the right main bronchus and act as ball valves, causing hyperinflation rather than collapse.

Mediastinal shift is away from the affected side in the ball-valve phase.

A button battery in the oesophagus is removed immediately, ideally within two hours.

Battery injury is electrolytic, generating hydroxide and causing liquefactive necrosis.

The double ring sign on the frontal film distinguishes a battery from a coin.

Honey or sucralfate may be given within 12 hours while awaiting removal, but never instead of it.

Unilateral foul nasal discharge in a child means a foreign body.

Grade severity before diagnosing cause in a child with stridor.

A quietening stridor with a falling respiratory rate signals impending arrest.

Croup is treated with a single dose of dexamethasone, commonly 0.6 mg/kg.

Nebulised adrenaline works within 30 minutes and wears off by two hours, so observe.

Epiglottitis is now rare after Hib vaccination but has not disappeared.

Never examine the throat, lie flat or cannulate a child with suspected epiglottitis.

Retropharyngeal abscess occurs under five, before the retropharyngeal nodes involute.

Quinsy is a disease of older children and adults, with trismus and uvular deviation.

Avoid amoxicillin where mononucleosis is possible.

Primary post-tonsillectomy haemorrhage is within 24 hours; secondary is at 5 to 10 days from infection.

Bilateral choanal atresia gives cyclical cyanosis relieved by crying, treated initially with an oral airway.

Laryngomalacia is the commonest infant stridor and usually resolves by 18 to 24 months.

Key formulas & results

Everything to memorise for the exam hall, in one card. Screenshot this for revision.

The organising tool
EVERY PAEDIATRIC ENT EMERGENCY IS A RACE AGAINST A LUMEN THAT WAS ALREADY SMALL. THE DIFFERENCE FROM ADULTS IS ALWAYS IN THE SAME DIRECTION: LESS RESERVE.
AIRWAY RESISTANCE RISES WITH THE FOURTH POWER OF RADIUS, OXYGEN RESERVE IS MINIMAL, AND CHILDREN CANNOT REPORT WHAT HAPPENED.
Why children desaturate in seconds
OXYGEN CONSUMPTION IS ROUGHLY TWICE THE ADULT RATE PER KILOGRAM WHILE FUNCTIONAL RESIDUAL CAPACITY IS PROPORTIONALLY SMALLER.
THE INTERVAL BETWEEN APNOEA AND DESATURATION IS MEASURED IN SECONDS, WHICH IS WHY PREOXYGENATION AND PREPARATION MATTER MORE THAN SPEED OF ACTION.
The foreign body rule
A CONVINCING CHOKING HISTORY IS AN INDICATION FOR RIGID BRONCHOSCOPY REGARDLESS OF A NORMAL CHEST RADIOGRAPH.
MOST PAEDIATRIC INHALED FOREIGN BODIES ARE ORGANIC AND RADIOLUCENT, SO THE FILM SHOWS ONLY SECONDARY EFFECTS. THE SYMPTOM-FREE INTERVAL IS EXPECTED AND FALSELY REASSURING.
The ball valve
AIR ENTERS PAST THE OBJECT ON INSPIRATION AS THE AIRWAY WIDENS AND IS TRAPPED ON EXPIRATION AS IT NARROWS, GIVING HYPERINFLATION RATHER THAN COLLAPSE.
THE FILM SHOWS A HYPERLUCENT LUNG, A DEPRESSED HEMIDIAPHRAGM AND MEDIASTINAL SHIFT AWAY FROM THE AFFECTED SIDE, BEST SEEN ON EXPIRATORY OR DECUBITUS VIEWS.
Why the right main bronchus
IT IS WIDER, SHORTER AND MORE VERTICAL THAN THE LEFT, AN ASYMMETRY THAT BECOMES MORE MARKED WITH AGE.
IN VERY YOUNG CHILDREN THE TWO SIDES ARE MORE SYMMETRICAL, SO LEFT-SIDED FOREIGN BODIES ARE PROPORTIONALLY COMMONER THAN IN ADULTS.
The choking algorithm by age
EFFECTIVE COUGH: DO NOT INTERFERE. INFANT UNDER 1 YEAR: FIVE BACK BLOWS THEN FIVE CHEST THRUSTS. CHILD OVER 1 YEAR: FIVE BACK BLOWS THEN FIVE ABDOMINAL THRUSTS.
ABDOMINAL THRUSTS ARE AVOIDED IN INFANTS BECAUSE THE LIVER IS RELATIVELY LARGE AND UNPROTECTED BY THE RIB CAGE. NEVER SWEEP THE MOUTH BLINDLY.
Button battery mechanism
NOT CHEMICAL LEAKAGE BUT ELECTROLYSIS. CURRENT FLOWS THROUGH MUCOSA, GENERATING HYDROXIDE AT THE NEGATIVE POLE, AND THE ALKALI PRODUCES LIQUEFACTIVE NECROSIS. INJURY BEGINS WITHIN TWO HOURS.
THIS IS WHY A DEAD BATTERY IS STILL DANGEROUS AND WHY THE TIMELINE IS MEASURED IN HOURS RATHER THAN DAYS.
Battery versus coin
FRONTAL FILM: DOUBLE RING OR HALO SIGN FOR A BATTERY, UNIFORM DISC FOR A COIN. LATERAL FILM: STEP-OFF AT THE EDGE FOR A BATTERY.
THE HISTORY IS UNRELIABLE, SO THE RADIOGRAPH DECIDES. A REPORTED COIN THAT LOOKS LIKE A BATTERY IS TREATED AS A BATTERY.
Honey and sucralfate
MAY BE GIVEN WHILE AWAITING ENDOSCOPY IN INGESTIONS OF UP TO 12 HOURS, TO COAT THE BATTERY AND SLOW LOCAL HYDROXIDE GENERATION. HONEY IN CHILDREN AGED 12 MONTHS AND OVER.
NEITHER SUBSTITUTES FOR REMOVAL AND NEITHER IS GIVEN BEYOND 12 HOURS OR IF PERFORATION IS SUSPECTED. THEY SLOW DAMAGE RATHER THAN PREVENTING IT.
Unilateral nasal discharge
UNILATERAL FOUL-SMELLING NASAL DISCHARGE IN A CHILD IS A FOREIGN BODY UNTIL PROVED OTHERWISE.
A NASAL BUTTON BATTERY IS AN EMERGENCY FOR THE SAME ELECTROLYTIC REASON AND CAN DESTROY THE SEPTUM WITHIN HOURS.
The deceptive improvement
A FALLING RESPIRATORY RATE WITH QUIETENING STRIDOR IN A TIRING CHILD INDICATES IMPENDING ARREST, NOT IMPROVEMENT.
REDUCED AIR MOVEMENT PRODUCES LESS NOISE. AGITATION, THEN DROWSINESS, THEN A SILENT CHEST IS THE SEQUENCE TO FEAR.
Croup versus epiglottitis
CROUP: 6 MONTHS TO 3 YEARS, GRADUAL AND NOCTURNAL, BARKING COUGH, HOARSE VOICE, NO DROOLING. EPIGLOTTITIS: RAPID OVER HOURS, TOXIC, DROOLING, MUFFLED VOICE, SITTING FORWARD, NO COUGH.
THE ABSENCE OF COUGH IN EPIGLOTTITIS AND ITS PROMINENCE IN CROUP IS ONE OF THE MOST RELIABLE DISCRIMINATORS.
Croup treatment
A SINGLE DOSE OF ORAL DEXAMETHASONE, COMMONLY 0.6 MG/KG, WITH EVIDENCE FOR 0.15 TO 0.3 MG/KG LESS CLEAR. NEBULISED ADRENALINE AROUND 400 MICROGRAMS/KG TO A MAXIMUM OF 5 MG IN MODERATE TO SEVERE DISEASE.
DEXAMETHASONE HELPS EVEN IN MILD DISEASE AND REDUCES RETURN VISITS. ADRENALINE WORKS BY 30 MINUTES AND WEARS OFF BY TWO HOURS, SO OBSERVE RATHER THAN DISCHARGE.
Epiglottitis: what not to do
DO NOT EXAMINE THE THROAT, DO NOT LIE THE CHILD FLAT, DO NOT ATTEMPT CANNULATION. KEEP UPRIGHT WITH A PARENT AND INSPECT AND INTUBATE IN THEATRE.
ANTIBIOTICS FOLLOW AIRWAY CONTROL. HIB VACCINATION CUT INCIDENCE IN UNDER-FIVES BY OVER 90 PER CENT, BUT THE DISEASE HAS NOT DISAPPEARED.
Retropharyngeal abscess
OCCURS UNDER ABOUT FIVE YEARS, BECAUSE THE RETROPHARYNGEAL LYMPH NODES DRAINING THE NASOPHARYNX INVOLUTE AFTER THAT AGE.
NECK STIFFNESS AND REFUSAL TO SWALLOW CAN MIMIC MENINGITIS. LATERAL NECK FILM SHOWS PREVERTEBRAL WIDENING; THE RISKS ARE AIRWAY OBSTRUCTION AND MEDIASTINAL SPREAD.
Quinsy
SEVERE UNILATERAL PAIN, TRISMUS, A MUFFLED HOT POTATO VOICE AND UVULAR DEVIATION AWAY FROM THE SWELLING. TREATED BY DRAINAGE PLUS ANTIBIOTICS.
IT IS A DISEASE OF OLDER CHILDREN AND ADULTS RATHER THAN TODDLERS, WHICH SEPARATES IT FROM RETROPHARYNGEAL ABSCESS BY AGE ALONE.
Post-tonsillectomy bleeding
PRIMARY WITHIN 24 HOURS IS SURGICAL AND USUALLY NEEDS THEATRE. SECONDARY AT 5 TO 10 DAYS IS FROM INFECTION AND SLOUGH SEPARATION AND IS USUALLY MANAGED MEDICALLY.
A CHILD WHO HAS SWALLOWED BLOOD MAY HAVE AN EMPTY MOUTH, SO TACHYCARDIA, PALLOR AND REPEATED SWALLOWING ARE MORE RELIABLE THAN VISIBLE BLEEDING.
Paediatric epistaxis
ALMOST ALL FROM LITTLE'S AREA, WHERE FIVE VESSELS ANASTOMOSE IN THE KIESSELBACH PLEXUS BENEATH THIN ANTERIOR SEPTAL MUCOSA.
PINCH THE SOFT CARTILAGINOUS NOSE, NOT THE BONY BRIDGE, FOR TEN UNINTERRUPTED MINUTES SITTING FORWARD. NEVER CAUTERISE BOTH SIDES OF THE SEPTUM AT ONE SITTING.
Corrosive injury by agent
ALKALI CAUSES LIQUEFACTIVE NECROSIS THAT PENETRATES DEEPLY AND CONTINUES AFTER CONTACT, INJURING THE OESOPHAGUS. ACID CAUSES COAGULATIVE NECROSIS WITH A LIMITING ESCHAR AND INJURES THE STOMACH MORE.
DO NOT INDUCE VOMITING, DO NOT NEUTRALISE, DO NOT PASS A BLIND NASOGASTRIC TUBE. ABSENCE OF ORAL BURNS DOES NOT EXCLUDE OESOPHAGEAL INJURY.
Choanal atresia
NEONATES ARE OBLIGATE NASAL BREATHERS, SO BILATERAL ATRESIA GIVES CYCLICAL CYANOSIS RELIEVED BY CRYING, WHICH FORCES MOUTH BREATHING.
FAILURE TO PASS A CATHETER THROUGH EACH NOSTRIL SUGGESTS IT AND CT CONFIRMS. AN ORAL AIRWAY IS THE IMMEDIATE TREATMENT, AND CHARGE SYNDROME MUST BE SOUGHT.
Laryngomalacia
THE COMMONEST CAUSE OF INFANT STRIDOR: INSPIRATORY, WORSE ON FEEDING, CRYING AND SUPINE, BETTER WHEN PRONE. APPEARS IN THE FIRST WEEKS, PEAKS AROUND SIX MONTHS, RESOLVES BY 18 TO 24 MONTHS.
MOST NEED ONLY REASSURANCE AND GROWTH MONITORING. FAILURE TO THRIVE, APNOEA OR SEVERE OBSTRUCTION INDICATES SUPRAGLOTTOPLASTY.
Acquired subglottic stenosis
MOST OFTEN FOLLOWS PROLONGED NEONATAL INTUBATION, GIVING BIPHASIC STRIDOR AND RECURRENT CROUP THAT DOES NOT BEHAVE LIKE CROUP.
RECURRENT CROUP IN AN INFANT WITH A HISTORY OF NEONATAL INTENSIVE CARE IS AN INDICATION FOR ENDOSCOPY RATHER THAN ANOTHER COURSE OF STEROIDS.
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Traps NEET PG sets — and how to dodge them

These are the exact option-traps and misreads that cost marks under negative marking.

WATCH OUT
Discharging a child with a choking history because the chest radiograph is normal
Most paediatric inhaled foreign bodies are organic and radiolucent, so the film can only show secondary effects such as air trapping. A convincing history mandates rigid bronchoscopy, because a retained peanut causes recurrent pneumonia, bronchiectasis or abscess over subsequent months.
WATCH OUT
Expecting collapse rather than hyperinflation on the film
A bronchial foreign body initially acts as a ball valve, admitting air on inspiration and trapping it on expiration. The affected side is hyperlucent with a depressed hemidiaphragm and mediastinal shift away from it, best demonstrated on an expiratory or decubitus view.
WATCH OUT
Performing abdominal thrusts on a choking infant
The infant liver is relatively large and poorly protected by the rib cage, so abdominal thrusts risk hepatic laceration. Under one year the sequence is five back blows followed by five chest thrusts, and blind finger sweeps are never performed at any age.
WATCH OUT
Treating a radio-opaque disc in the oesophagus as a coin
A double ring or halo sign on the frontal film and a step-off on the lateral identify a button battery, and the parental history is unreliable. Any doubt is resolved in favour of treating it as a battery, because the consequence of delay is liquefactive necrosis.
WATCH OUT
Delaying battery removal to give honey or sucralfate
These agents coat the battery and slow hydroxide generation but do not prevent injury, and they are adjuncts within the first 12 hours only. Nothing should postpone endoscopic removal, which is targeted at under two hours from ingestion.
WATCH OUT
Giving honey or sucralfate for a battery ingested more than 12 hours earlier
Beyond 12 hours the mucosa may already be perforated, and introducing viscous material into a perforated oesophagus is harmful. The same applies where perforation is clinically suspected at any time point.
WATCH OUT
Treating unilateral purulent nasal discharge in a child as sinusitis
One-sided foul discharge in an otherwise well child is a retained foreign body until proved otherwise. Removal is attempted only with adequate immobilisation and instruments, since posterior displacement can convert a nasal problem into an inhaled one.
WATCH OUT
Reading a quietening stridor as clinical improvement
In a tiring child, less noise means less air movement rather than less obstruction, and a falling respiratory rate with drowsiness is the pre-arrest state. Severity is graded on work of breathing and conscious level, not on audibility.
WATCH OUT
Discharging a child soon after nebulised adrenaline
Adrenaline improves symptom scores within 30 minutes but the effect wears off after about two hours, so rebound is common. A child who required it is observed for a period after the dose rather than discharged on the immediate improvement.
WATCH OUT
Withholding dexamethasone in mild croup
A single oral dose, commonly 0.6 mg/kg, benefits even mild disease and reduces return visits and admission. Reserving steroid for severe cases forfeits the intervention with the strongest evidence base in the condition.
WATCH OUT
Assuming epiglottitis no longer occurs after Hib vaccination
Incidence in under-fives fell by more than 90 per cent, but the disease persists in unvaccinated children, in vaccine failures and with non-typeable and other organisms. Vaccination status reduces the probability without excluding the diagnosis.
WATCH OUT
Attributing neck stiffness in a toddler to meningitis alone
Retropharyngeal abscess in a child under five produces neck stiffness, refusal to swallow and refusal to move the neck, and is regularly mistaken for meningitis. A lateral neck radiograph showing prevertebral widening and contrast CT establish it.
WATCH OUT
Prescribing amoxicillin for exudative tonsillitis in an adolescent
Infectious mononucleosis is common in this group and aminopenicillins produce a florid maculopapular rash. Where mononucleosis is plausible, choose a different agent and consider testing before treating.
WATCH OUT
Judging post-tonsillectomy bleeding by what is visible in the mouth
Children swallow blood, so the oropharynx can look deceptively clean while significant loss continues. Tachycardia, pallor and repeated swallowing are the reliable signs, and the timing separates a primary surgical bleed from a secondary infective one.
WATCH OUT
Pinching the bony bridge of the nose for epistaxis
Almost all paediatric bleeding is from Little's area on the anterior septum, which is compressed only by pinching the soft cartilaginous part. Pressure on the bony bridge achieves nothing, and the child should sit forward rather than tilt back.
WATCH OUT
Attempting to neutralise a swallowed corrosive
Neutralisation is exothermic and adds a thermal burn to a chemical one, and inducing vomiting re-exposes the oesophagus to the agent. Blind nasogastric intubation risks perforation. Endoscopy within 24 to 48 hours grades the injury and predicts stricture.

Exam-pattern practice

PYQ-style questions with full solutions. Work through them as a readiness check — mark yourself honestly and get your gap report at the end.

Readiness check

Are you exam-ready for "Pediatric ENT Emergencies"?

9 problems from this chapter. Try each one, reveal the worked solution, mark yourself honestly — get your gap report at the end.

9 questions~6 min

5-minute revision

The whole chapter, distilled. Read this the night before the exam.

  • Paediatric emergencies differ by having less reserve, not different diseases.
  • Airway resistance rises with the fourth power of the radius.
  • Children consume twice the adult oxygen per kilogram with a smaller FRC.
  • Deterioration is abrupt rather than gradual.
  • Toddlers dominate inhaled foreign body statistics.
  • The right main bronchus is wider, shorter and more vertical.
  • A symptom-free interval after choking is expected and misleading.
  • Most inhaled objects in children are organic and radiolucent.
  • A foreign body acts as a ball valve giving hyperinflation.
  • Mediastinal shift is away from the affected side.
  • Expiratory and decubitus films demonstrate air trapping.
  • A normal chest radiograph never excludes an inhaled foreign body.
  • Rigid bronchoscopy is diagnostic and therapeutic.
  • An effective cough is left alone.
  • Infants under 1 year: five back blows then five chest thrusts.
  • Children over 1 year: five back blows then five abdominal thrusts.
  • Never perform a blind finger sweep.
  • Button battery injury is electrolytic, not chemical leakage.
  • Hydroxide at the negative pole causes liquefactive necrosis.
  • Injury begins within two hours.
  • Double ring on the frontal film means battery.
  • Step-off on the lateral film means battery.
  • Remove an oesophageal battery immediately, ideally within 2 hours.
  • Honey or sucralfate may be given within 12 hours only.
  • Honey is used from 12 months of age.
  • Neither agent substitutes for removal.
  • Late battery complications include stricture and fistula.
  • Aorto-oesophageal fistula can present with a sentinel bleed.
  • Unilateral foul nasal discharge in a child means a foreign body.
  • A nasal battery can destroy the septum within hours.
  • Grade stridor severity before diagnosing the cause.
  • Quietening stridor with a falling rate means impending arrest.
  • Croup: barking cough, hoarse voice, no drooling.
  • Epiglottitis: toxic, drooling, muffled voice, no cough.
  • Bacterial tracheitis is toxic with thick purulent secretions.
  • Croup is treated with a single dexamethasone dose, commonly 0.6 mg/kg.
  • Dexamethasone helps even in mild croup.
  • Nebulised adrenaline is 400 micrograms/kg to a maximum of 5 mg.
  • Adrenaline works by 30 minutes and wears off by two hours.
  • Hib vaccination cut epiglottitis in under-fives by over 90 per cent.
  • Epiglottitis still occurs in the unvaccinated and in vaccine failures.
  • Never examine, lie flat or cannulate a suspected epiglottitis.
  • Antibiotics follow airway control in epiglottitis.
  • Retropharyngeal abscess occurs under five years.
  • Retropharyngeal nodes involute after about age five.
  • Prevertebral widening on lateral neck film supports the diagnosis.
  • Mediastinal spread is the feared complication.
  • Centor criteria estimate streptococcal likelihood.
  • Quinsy affects older children and adults with trismus.
  • The uvula deviates away from a peritonsillar swelling.
  • Avoid amoxicillin where mononucleosis is possible.
  • Primary post-tonsillectomy bleeding is within 24 hours and surgical.
  • Secondary bleeding is at 5 to 10 days and infective.
  • A swallowed bleed can leave a deceptively clean mouth.
  • Paediatric epistaxis comes from Little's area.
  • Pinch the cartilage, not the bony bridge, sitting forward.
  • Never cauterise both septal sides at one sitting.
  • Adolescent boy with heavy epistaxis: think angiofibroma, do not biopsy.
  • Alkali causes deep liquefactive oesophageal injury.
  • Acid causes coagulative necrosis and injures the stomach more.
  • Do not induce vomiting, neutralise, or pass a blind tube.
  • Absence of oral burns does not exclude oesophageal injury.
  • Neonates are obligate nasal breathers.
  • Bilateral choanal atresia gives cyclical cyanosis relieved by crying.
  • An oral airway is the immediate treatment.
  • Choanal atresia is associated with CHARGE syndrome.
  • Laryngomalacia is the commonest infant stridor.
  • It is better prone and worse supine, and resolves by 18 to 24 months.
  • Acquired subglottic stenosis follows prolonged neonatal intubation.
  • Recurrent croup after neonatal intensive care needs endoscopy.

NEET PG question blueprint

How this topic is asked, tier by tier — so you can prep to the pattern.

Typical weightage: Each NEET PG question is worth +4/-1; paediatric ENT emergencies contribute 4-6 questions per attempt and overlap with Paediatrics, Anaesthesia and Forensic Medicine

Question styleMarks eachTypical countWhat it tests
Inhaled foreign body4~1The ball-valve mechanism, radiographic findings and the primacy of the choking history
Button battery4~1Electrolytic mechanism, radiographic discrimination, removal timing and adjunct limits
Croup and epiglottitis4~2Discriminating features, dexamethasone and adrenaline, and the contraindicated actions
Stridor severity4~1Recognising exhaustion masquerading as improvement and escalating appropriately
Neonatal airway4~1Choanal atresia, laryngomalacia and acquired subglottic stenosis
Post-tonsillectomy bleeding4~1Primary versus secondary timing, cause and management
Corrosive ingestion4~1Alkali versus acid injury patterns and the three contraindicated interventions

Exam-hall strategy

Battle-tested tips from mentors and toppers for this topic under the sectional clock.

  1. Look for a choking history in the stem; it outranks any imaging finding offered.
  2. Check whether a swallowed disc is described as having a ring or a step-off.
  3. For stridor stems, note whether the child is drooling and whether there is a cough.
  4. Read trends in respiratory rate rather than absolute values in a tiring child.
  5. Check the age before choosing between retropharyngeal abscess and quinsy.
  6. For post-tonsillectomy bleeding, the day number decides the answer.
  7. In a neonate, cyclical cyanosis relieved by crying is always choanal atresia.
  8. With NEET PG's +4/-1 marking, the croup versus epiglottitis table, the battery timings and the choking algorithm are high-certainty recall worth banking early.
  9. Under the 5-group, 42-minute time-bound format, clear those fast and spend the remaining time on the stridor severity and corrosive ingestion stems, since a closed group cannot be reopened.

Beyond the exam

Where this skill shows up in the job you're competing for — and in life.

Booking the bronchoscopy on the history

Acting on a parent's account of choking rather than on a reassuring film is what prevents the child returning months later with bronchiectasis in one lobe.

Reading the disc before calling it a coin

Checking a swallowed disc for a double ring on the frontal film and a step-off on the lateral takes seconds and separates a routine ingestion from a two-hour emergency.

Escalating on the falling respiratory rate

Treating a quietening child as deteriorating rather than settling is the observation that gets the anaesthetist to the bedside before the arrest rather than after it.

Leaving the throat alone

Not examining a drooling, toxic child with stridor is an active clinical decision, and it keeps a marginal airway open until it can be secured in theatre.

Where else this topic is tested

Prepare once, score in every exam that asks it.

FMGE / NExTVery high overlap — foreign body management, croup, epiglottitis and choanal atresia are examined at identical depth
USMLE Step 2 CKHigh overlap — button battery ingestion, croup and airway emergencies are shared, with more emphasis on emergency department disposition
MD Paediatrics and MS ENT entranceFoundational — assumed working knowledge, with bronchoscopy technique, airway reconstruction and paediatric anaesthesia examined far more deeply

Questions aspirants ask

Pulled from the Q&A community and mentor sessions.

Because the film is not looking at the object, it is looking for the object's consequences, and those consequences take time and the right technique to appear. The great majority of foreign bodies inhaled by toddlers are organic, most commonly peanuts, seeds and other food fragments, and organic material is radiolucent, so it is simply invisible on a plain film. What the radiograph can show is air trapping distal to a partially obstructing object, which produces a hyperlucent lung, a depressed hemidiaphragm and mediastinal shift away from the affected side. But that pattern requires the object to be acting as a ball valve at the time of the film, and it is far more visible on an expiratory film or a lateral decubitus view than on the standard inspiratory film that a distressed toddler is most likely to receive. Reported sensitivity of plain radiography in this setting is therefore poor, with a substantial proportion of proven foreign bodies having entirely normal films. The consequence of relying on the film is not a delayed diagnosis but a different disease: the object remains, provokes granulation and inflammation, and the child returns weeks or months later with recurrent pneumonia in the same lobe, bronchiectasis or a lung abscess, at which point removal is technically much harder. A convincing choking history is therefore the indication for bronchoscopy, and the radiograph can only add information, never subtract it.

Because a coin causes injury only by pressure, whereas a battery actively generates a chemical burn from the moment it makes contact. The mechanism is often misunderstood as leakage of battery contents, which would at least require a damaged casing. What actually happens is electrolysis. When the battery lodges against moist mucosa, the tissue completes an external circuit between the two poles, and current flows through the mucosa itself. At the negative pole this electrolyses water, generating hydroxide ions and therefore a strongly alkaline environment right at the tissue surface. Alkali produces liquefactive necrosis, which penetrates progressively rather than self-limiting, so the injury deepens for as long as the battery remains and continues for a period after removal. Experimental and clinical data place the onset of significant mucosal damage within two hours, which is why the target for removal is measured in hours rather than the day or two acceptable for a lodged coin. Two corollaries follow. A battery that is flat and no longer powers a toy still carries enough residual voltage to do this, so exhausted batteries are not safe. And because the burn is directional, deepest at the negative pole, knowing the orientation from the lateral radiograph predicts which structures are at risk, whether that is the trachea anteriorly or the aorta posteriorly.

Because stridor is generated by turbulent airflow, so producing it requires both a narrowed airway and enough air moving through it. A child with severe obstruction who is still moving reasonable volumes of air makes a great deal of noise, and that noise, uncomfortable as it is to listen to, is evidence of ongoing ventilation. As the child tires, tidal volume falls and respiratory muscles fatigue, so less air passes through the same narrowing and the stridor becomes quieter. Nothing about the obstruction has improved. The accompanying signs make the direction of travel clear if they are looked for. The respiratory rate falls rather than rises, because exhausted intercostal and diaphragmatic muscles cannot sustain a high rate. Recession may paradoxically decrease for the same reason. Carbon dioxide accumulates, producing first agitation and then drowsiness, and the drowsiness is regularly mistaken for a child settling to sleep. The endpoint is a silent chest and respiratory arrest. This is why severity in paediatric stridor is graded on work of breathing, conscious level, oxygen saturation and heart rate rather than on how loud the noise is, and why an apparently improving child whose respiratory rate is falling should trigger escalation rather than reassurance.

Because the anatomical structure that becomes infected exists only in early childhood. The retropharyngeal space lies between the posterior pharyngeal wall and the prevertebral fascia, and in young children it contains two chains of lymph nodes that drain the nasopharynx, adenoids, posterior paranasal sinuses and middle ear. When any of those sites is infected, organisms track to these nodes, which suppurate and form an abscess in the space. These nodes involute progressively and have usually disappeared by around five years of age, which removes the mechanism. After that age retropharyngeal collections do still occur, but they arise differently, from direct trauma such as a fall with an object in the mouth, from instrumentation, or from spread of vertebral osteomyelitis, and they are much less common. The clinical presentation follows the anatomy too. The child holds the neck stiff and resists movement, because moving the neck stretches the inflamed prevertebral tissues, which is why the picture is regularly mistaken for meningitis. Swallowing is painful, drooling may occur, and the airway can be compromised by anterior displacement of the pharyngeal wall. The space communicates inferiorly with the mediastinum, so untreated infection can descend, which is the complication that makes early recognition and drainage important.

Because the two produce different kinds of necrosis, and the kind of necrosis determines both how deep the injury goes and how long the agent lingers at each site. Alkali causes liquefactive necrosis. It saponifies fats and denatures proteins in a way that dissolves the tissue barrier rather than sealing it, so the agent keeps penetrating and the injury continues to deepen for some time after contact ends. Because it destroys tissue as it goes, it does not provoke the immediate intense pain that would stop swallowing, and it dwells in the oesophagus, which is why alkali is classically the oesophageal injury and why full-thickness damage with perforation and later stricture is common. Acid causes coagulative necrosis. It precipitates proteins into a firm eschar, and that eschar physically limits further penetration, so injury tends to be shallower at any given site. Acid also causes immediate severe pain, so it is often swallowed quickly and passes into the stomach, where it pools in the antrum and produces the more severe gastric injury characteristic of acid ingestion. Both share the same management errors to avoid: not inducing vomiting, which re-exposes the oesophagus; not attempting neutralisation, which releases heat and adds a thermal burn; and not passing a blind nasogastric tube through a friable oesophagus.
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